Unstable blood pressure after sympathectomy
Risks
Side effects of sympathectomy may include decreased blood pressure while standing, which may cause fainting spells. After sympathectomy in men, semen is sometimes ejaculated into the bladder, which may impair fertility. After a sympathectomy done by inserting an endoscope in the chest cavity, patients may experience chest pain with deep breathing. This problem usually disappears within two weeks. They may also experience pneumothorax (air in the chest cavity).
Sympathectomy suppresses baroreflex control of heart rate
Endoscopic Thoracic Sympathectomy Suppresses Baroreflex Control of Heart Rate in Patients with Essential Hyperhidrosis
2004 International Anesthesia Research Society
Australian Story 3!
TIMBO McCarty, an Australian/New Zealander who has been suffering from the side-effects of ETS for eight years and has been working on this website alongside me as well as helping organise meetings, had enough of the side-effects and committed suicide on October 29 at the age of 45. His Father and Mother asked me to put this on the website and to let everyone know that he had had enough. Timbo was always there for me as a friend to talk when I felt everything was getting to me. Timbo was also was a fighter. He wanted ETS surgery to be stopped and was depressed that the surgery is still being performed routinely. He was aghast that his surgeon, Dr Lin in Taiwan, who performed ESB (clamping of the T2 nerve), removed his clamps at a fee yet still performed reversals routinely even though so many people have come forward with complaints.....
This is the second suicide in two years from people who have had terrible side-effects.
Support groups for people who had sympathectomy and ended up with disabling side-effects
World Against Sympathectomy | http://www.truthaboutets.com/ |
The Sympathetic Association | http://home.swipnet.se/sympatiska/index3.htm |
Patients Against Sympathetic Surgery | http://www.ets-sideeffects.netfirms.com/ |
Radisson Group | http://www.noetsuk.com/ |
Family of Compensatory Sweating Sufferers | http://home.pchome.com.tw/family/vivi12175/ |
ETS Side Effects | http://www.geocities.jp/etscontroversialop/index.html |
Hyperhidrosis Forum | http://www.terra.es/personal8/hiperhidrosis/principal.htm |
Sympathectomy is prohibited for patients under 20 years in Taiwan. Why?
|   | |||
| Min-Huei Hsu Department of Neurourgery, Taipei City Hospital, Zhongxiao Branch Send letter to journal: Email Min-Huei Hsu
| Endoscopic thoracic sympathectomy (ETS) has come into widespread use for palmar hyperhidrosis. Side sffects after ETS was widely discussed in Taiwan society in the past few months. Lots of people in Taiwan suffer from hyperhidrosis palmaris. ETS is covered by the National Health Insurance, and patient billing for this operation does not exceed US$ 60. This is why this operation is so popular here 1 . Patients with severe compensatory sweating after ETS must change clothes several times a day (some patients complained that they change as often as 10 times a day), resulting in serious impact on work and social interaction. Patients suffering from such serious side effects in Taiwan have formed a support group based on an Internet discussion forum to request the government to take this problem seriously. Starting in October 2004, The Department of Health, Executive Yuan, Taiwan, has prohibited surgeons from performing this operation on patients under 20 years old. To our knowledge, this type of support group also exists in United States, England, Sweden, Spain and Japan (Table 1). ETS is a relatively safe and simple procedure. However the side effects are possibly devastating 2 . All physicians providing this service and all peoples preparing to undergo this treatment should know this well. | |||
calcium accumulation following sympathectomy
Developmental status of sympathetic innervation in relation to calcium accumulation by submandibular gland following reserpine, surgical sympathectomy or cyclocytidine
CA Schneyer and JH YuProceedings of the Society for Experimental Biology and Medicine, Vol 179, 143-146, Copyright © 1985 by Society for Experimental Biology and Medicine
number of hemorrhages in the denervated half of the brain...
Sh. S. Tashaev
Received: 17 March 1980
The number of hemorrhages in the denervated half of the brain, which was twice that. in the intact half, is evidence that the sympathetic innervation of the ...
www.springerlink.com/index/T8J46575171L3G34.pdf -
Without Abstract
Key Words adaptation - hemorrhage - hypotension - hypertension - reinfusion
sympathetic innervation, adrenergic receptors, and a possible local catecholamine production in the development of patellar tendinopathy (tendinosis)
Studies on the importance of sympathetic innervation, adrenergic receptors, and a possible local catecholamine production in the development of patellar tendinopathy (tendinosis) in man |
| Patrik Danielson 1 *, Håkan Alfredson 2, Sture Forsgren 1 |
| 1Department of Integrative Medical Biology, Section for Anatomy, Umeå University, Umeå, Sweden 2Department of Surgical and Perioperative Science, Sports Medicine, Umeå University, Umeå, Sweden |
| email: Patrik Danielson (patrik.danielson@anatomy.umu.se) |
*Correspondence to Patrik Danielson, Department of Integrative Medical Biology, Section for Anatomy, Umeå University, SE-901 87 Umeå, Sweden
Funded by:
Faculty of Medicine at Umeå University The Swedish National Centre for Research in Sports The County Council of Västerbotten The Arnerska Research Foundation
| Keywords |
tyrosine hydroxylase •  (alpha)-1 • (alpha)-2A •  (beta)-1 • adrenoreceptors • tenocytes |
| Abstract |
| Changes in the patterns of production and in the effects of signal substances may be involved in the development of tendinosis, a chronic condition of pain in human tendons. There is no previous information concerning the patterns of sympathetic innervation in the human patellar tendon. In this study, biopsies of normal and tendinosis patellar tendons were investigated with immunohistochemical methods, including the use of antibodies against tyrosine hydroxylase (TH) and neuropeptide Y, and against 1-, 2A-, and 1-adrenoreceptors. It was noticed that most of the sympathetic innervation was detected in the walls of the blood vessels entering the tendon through the paratendinous tissue, and that the tendon tissue proper of the normal and tendinosis tendons was very scarcely innervated. Immunoreactions for adrenergic receptors were noticed in nerve fascicles containing both sensory and sympathetic nerve fibers. High levels of these receptors were also detected in the blood vessel walls; 1-adrenoreceptor immunoreactions being clearly more pronounced in the tendinosis tendons than in the tendons of controls. Interestingly, immunoreactions for adrenergic receptors and TH were noted for the tendon cells (tenocytes), especially in tendinosis tendons. The findings give a morphological correlate for the occurrence of sympathetically mediated effects in the patellar tendon and autocrine/paracrine catecholamine mechanisms for the tenocytes, particularly, in tendinosis. The observation of adrenergic receptors on tenocytes is interesting, as stimulation of these receptors can lead to cell proliferation, degeneration, and apoptosis, events which are all known to occur in tendinosis. Furthermore, the results imply that a possible source of catecholamine production might be the tenocytes themselves. Microsc. Res. Tech., 2007. © 2007 Wiley-Liss, Inc. |
Received: 4 July 2006; Accepted: 26 October 2006
If you are wondering why did you gain weight since your sympathectomy....
: Am J Physiol Regul Integr Comp Physiol. 2004 Jun;286(6):R1167-75.
Sympathetic innervation of white adipose tissue and its regulation of fat cell number.
Molecular and Cellular Biology and Pathobiology Program, Medical University of South Carolina, Charleston, South Carolina 29425, USA.
White adipose tissue (WAT) is innervated by the sympathetic nervous system (SNS), and the central origins of this innervation have been demonstrated for inguinal and epididymal WAT (iWAT and eWAT, respectively) using a viral transneuronal tract tracer, the pseudorabies virus (PRV). Although the more established role of this sympathetic innervation of WAT is as a major stimulator of lipid mobilization, this innervation also inhibits WAT fat cell number (FCN); thus, local denervation of WAT leads to marked increases in WAT mass and FCN. The purpose of this study was to extend our understanding of the SNS regulation of FCN using neuroanatomical and functional analyses. Therefore, we injected PRV into retroperitoneal WAT (rWAT) to compare the SNS outflow to this pad from what already is known for iWAT and eWAT. In addition, we tested the ability of local unilateral denervation of rWAT or iWAT to promote increases in WAT mass and FCN vs. their contralateral neurally intact counterparts. Although the overall pattern of innervation was more similar than different for rWAT vs. iWAT or eWAT, its SNS outflow appeared to involve more neurons in the suprachiasmatic and solitary tract nuclei. Denervation produced significant increases in WAT mass and FCN for both iWAT and rWAT, but FCN was increased significantly more in iWAT than in rWAT. These data suggest differences in origins of the sympathetic outflow to WAT and functional differences in the WAT SNS innervation that could contribute to the differential propensity for fat cell proliferation across WAT depots in vivo.
The nervous system and adipose tissue
THE NERVOUS SYSTEM AND ADIPOSE TISSUEKatharine Dalziel, MD, MBBS, MRCP
1989
...whereas sympathectomy abolishes the
vasoconstrictor reaction, indicating that it is mediated by a local sympathetic axon
reflex.
Extraneous agents, such as large doses of alcohol and morphine, cause increased
lipolysis. This action can be blocked by chemical sympathectomy and is believed to be
due to stimulation of the sympathetic system within the central nervous system.9
Thus the control of metabolism in white fat is complex and dependent on many factors,
both within the adipocytes themselves and in the organism as a whole.
Total chemical sympathectomy in experimental
animals results in loss of shivering and nonshivering thermogenesis, and death within a
few hours.9
Since the effects of sympathectomy on the denervated
area are profound with increased blood flow and warmth and decreased sweating, it is
possible that pain relief is secondary to these phenomena without requiring any sort of
aberrant neural conduction.
This has occurred after stellate ganglion
block37 and lumbar sympathectomy.38 McCallurn and Glynn37 propose that increased
activity in the sympathetic nerves as the effects of the anesthetic block lessen cause
pain, particularly in circumstances where there was abnormal sympathetic activity
before the procedure (such as sympathetic dystrophy). This type of pain is often
resistant to opioid analgesics but may respond well to transcutaneous nerve stimulation.
A similar reduction of fat
mobilization from fat depots occurs after VMH lesions, as after local sympathectomy,
suggesting that the sympathetic pathway to the adipose tissue runs through the VMH.71
These hypothalamic control areas are themselves sensitive to feedback mechanisms.
